The herpes simplex virus VP16-induced complex: the makings of a regulatory switch

This publication has 75 references indexed in Scilit:

• The VP16 Activation Domain Interacts with Multiple Transcriptional Components as Determined by Protein-Protein Cross-linking in Vivo
  Journal of Biological Chemistry, 2002
• Identification of an octamer element required for in vivo expression of the TIE1 gene in endothelial cells
  Biochemical Journal, 2001
• Loss of HCF-1–Chromatin Association Precedes Temperature-Induced Growth Arrest of tsBN67 Cells
  Molecular and Cellular Biology, 2001
• Transcription factors Oct-1 and NF-YA regulate the p53-independent induction of the GADD45 following DNA damage
  Oncogene, 2001
• Developmental and Cell-Cycle Regulation of Caenorhabditis elegans HCF Phosphorylation
  Biochemistry, 2001
• POU Domain Factors in the Neuroendocrine System: Lessons from Developmental Biology Provide Insights into Human Disease
  Endocrine Reviews, 2001
• The virtuoso of versatility: POU proteins that flex to fit 1 1Edited by P. Wright
  Journal of Molecular Biology, 2000
• A single-point mutation in HCF causes temperature-sensitive cell-cycle arrest and disrupts VP16 function.
  Genes & Development, 1997
• Differential positive control by Oct-1 and Oct-2: activation of a transcriptionally silent motif through Oct-1 and VP16 corecruitment.
  Genes & Development, 1993
• Identification of herpes simplex virus DNA sequences which encode a trans-acting polypeptide responsible for stimulation of immediate early transcription
  Journal of Molecular Biology, 1984