Specific N- or C-terminus modified dynorphin and β-endorphin peptides can selectively block excitatory opioid receptor functions in sensory neurons and unmask potent inhibitory effects of opioid agonists
- 27 February 1995
- journal article
- Published by Elsevier in Brain Research
- Vol. 673 (1) , 30-38
- https://doi.org/10.1016/0006-8993(94)01380-z
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Inhibition of the antianalgesic action of dynorphin A in mice by cholera toxinPharmacology Biochemistry and Behavior, 1993
- After chronic opioid exposure sensory neurons become supersensitive to the excitatory effects of opioid agonists and antagonists as occurs after acute elevation of GM1 gangliosideBrain Research, 1992
- Effects of morphine treatment on pro-opiomelanocortin systems in rat brainBrain Research, 1990
- Opioids can evoke direct receptor-mediated excitatory effects on sensory neuronsTrends in Pharmacological Sciences, 1990
- Maturation of opioid sensitivity of fetal mouse dorsal root ganglion neuron perikarya in organotypic cultures: Regulation by spinal cordNeuroscience, 1986
- Endogenous Opioids: Biology and FunctionAnnual Review of Neuroscience, 1984
- Opiate binding properties of naturally occurring N- and C-Terminus modified beta-endorphinsPeptides, 1981
- Influence of N-terminal acetylation and C-terminal proteolysis on the analgesic activity of β-endorphinBiochemical Journal, 1980
- Methionine-enkephalin antagonism and endorphin potentiation of narcotic-induced analgesiaEuropean Journal of Pharmacology, 1980
- Enhanced afferent synaptic functions in fetal mouse spinal cord-sensory ganglion explants following NGF-induced ganglion hypertrophyBrain Research, 1974