Tyrosine Phosphorylation of IκBα Activates NFκB through a Redox-regulated and c-Src-dependent Mechanism Following Hypoxia/Reoxygenation
Open Access
- 1 January 2003
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 278 (3) , 2072-2080
- https://doi.org/10.1074/jbc.m206718200
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Temporal pattern of NFκB activation influences apoptotic cell fate in a stimuli-dependent fashionJournal of Cell Science, 2002
- GPx-1 Gene Delivery Modulates NFκB Activation Following Diverse Environmental Injuries Through a Specific Subunit of the IKK ComplexAntioxidants and Redox Signaling, 2001
- Evidence for a Functional Association between Phosphatidylinositol 3-Kinase and c-src in the Spreading Response of Osteoclasts to Colony-Stimulating Factor-1Endocrinology, 2000
- Src family kinases are required for integrin but not PDGFR signal transductionThe EMBO Journal, 1999
- Redox-Mediated Gene Therapies for Environmental Injury: Approaches and ConceptsAntioxidants and Redox Signaling, 1999
- NFkappaB prevents apoptosis and liver dysfunction during liver regeneration.Journal of Clinical Investigation, 1998
- I kappa B alpha degradation is not a requirement for the X-ray-induced activation of nuclear factor kappa B in normal rat astrocytes and human brain tumour cellsInternational Journal of Radiation Biology, 1998
- Site-specific Tyrosine Phosphorylation of IκBα Negatively Regulates Its Inducible Phosphorylation and DegradationJournal of Biological Chemistry, 1996
- Hypoxia and Hypoxia/Reoxygenation Activate Src Family Tyrosine Kinases and p21rasin Cultured Rat Cardiac MyocytesBiochemical and Biophysical Research Communications, 1996
- Direct gene transfer of human CFTR into human bronchial epithelia of xenografts with E1–deleted adenovirusesNature Genetics, 1993