Abstract
The precipitate rigor mortis observed in rabbits after hypoglycaemic convulsions or after prolonged thyroid feeding is not due to accumulation of lactic acid, nor to increased acidity of the muscle, but to absence of glycogen and decrease in lactacidogen of the muscle. The appearance of rigor mortis is accelerated by decrease of glycogen in the muscles. Rigor mortis is immediate when the glycogen is exhausted, and death from complete hypoglycemia occurs. The mechanism of normal rigor mortis is discussed.

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