Disruption of tumor necrosis factor-α gene diminishes the development of atherosclerosis in ApoE-deficient mice
- 1 May 2005
- journal article
- Published by Elsevier in Atherosclerosis
- Vol. 180 (1) , 11-17
- https://doi.org/10.1016/j.atherosclerosis.2004.11.016
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- Exclusive expression of transmembrane TNF-α in mice reduces the inflammatory response in early lipid lesions of aortic sinusAtherosclerosis, 2004
- IL-1β AND TNF-α SUPPRESS APOLIPOPROTEIN (APO) E SECRETION AND APO A-I EXPRESSION IN HepG2 CELLSCytokine, 1998
- A role for macrophage scavenger receptors in atherosclerosis and susceptibility to infectionNature, 1997
- Accelerated Atherosclerosis in Mice Lacking Tumor Necrosis Factor Receptor p55Journal of Biological Chemistry, 1996
- Inhibition of Macrophage Scavenger Receptor Activity by Tumor Necrosis Factor-α Is Transcriptionally and Post-transcriptionally RegulatedJournal of Biological Chemistry, 1996
- Regulation of smooth muscle cell scavenger receptor expression in vivo by atherogenic diets and in vitro by cytokines.Journal of Clinical Investigation, 1995
- Atherosclerosis in mice lacking apo E. Evaluation of lesional development and progression.Arteriosclerosis and Thrombosis: A Journal of Vascular Biology, 1994
- The pathogenesis of atherosclerosis: a perspective for the 1990sNature, 1993
- Spontaneous Hypercholesterolemia and Arterial Lesions in Mice Lacking Apolipoprotein EScience, 1992
- Direct expression cloning of vascular cell adhesion molecule 1, a cytokine-induced endothelial protein that binds to lymphocytesCell, 1989