Delayed onset of huntington′s disease in mice in an enriched environment correlates with delayed loss of cannabinoid CB1 receptors
- 27 November 2003
- journal article
- research article
- Published by Elsevier in Neuroscience
- Vol. 123 (1) , 207-212
- https://doi.org/10.1016/s0306-4522(03)00595-5
Abstract
No abstract availableKeywords
This publication has 42 references indexed in Scilit:
- Endocannabinoids facilitate the induction of LTP in the hippocampusNature Neuroscience, 2002
- Transgenic models of Huntington'sdiseasePhilosophical Transactions Of The Royal Society B-Biological Sciences, 1999
- Cannabinoids decrease excitatory synaptic transmission and impair long‐term depression in rat cerebellar Purkinje cellsThe Journal of Physiology, 1998
- Exon 1 of the HD Gene with an Expanded CAG Repeat Is Sufficient to Cause a Progressive Neurological Phenotype in Transgenic MiceCell, 1996
- Expression of neurotrophin-3 mRNA in the rat visual cortex and hippocampus is influenced by environmental conditionsNeuroscience Letters, 1996
- Environment, Social Interaction, and Physical Activity as Determinants of Functional Outcome after Cerebral Infarction in the RatExperimental Neurology, 1996
- Risk Factors for Dementia in Parkinson's Disease: Effect of EducationNeuroepidemiology, 1995
- Environment Influences Functional Outcome of Cerebral Infarction in RatsStroke, 1995
- Physical activity enhances spatial learning performance with an associated alteration in hippocampal protein kinase C activity in C57BL/6 and DBA/2 miceBrain Research, 1993
- A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomesCell, 1993