Selective Degradation of the PKC-ϵ Isoform during Cell Death in AKR-2B Fibroblasts
- 1 May 2001
- journal article
- Published by Elsevier in Experimental Cell Research
- Vol. 266 (1) , 64-73
- https://doi.org/10.1006/excr.2001.5211
Abstract
No abstract availableKeywords
This publication has 52 references indexed in Scilit:
- Proteolytic cleavage of protein kinase Cμ upon induction of apoptosis in U937 cellsFEBS Letters, 1999
- Proteolytic Activation of Protein Kinase C δ and ϵ by Caspase-3 in U937 Cells During Chemotherapeutic Agent-Induced ApoptosisCellular Signalling, 1999
- Emerging roles of caspase-3 in apoptosisCell Death & Differentiation, 1999
- Targeting of Protein Kinase Cα to CaveolaeThe Journal of cell biology, 1998
- The Proteolytic Cleavage of Protein Kinase C Isotypes, Which Generates Kinase and Regulatory Fragments, Correlates with Fas‐Mediated and 12‐O‐Tetradecanoyl‐Phorbol‐13‐Acetate‐Induced ApoptosisEuropean Journal of Biochemistry, 1997
- Platelet‐derived growth factor isoforms prevent cell death during starvation of AKR‐2B fibroblastsJournal of Cellular Physiology, 1994
- Protein kinase C - a question of specificityTrends in Biochemical Sciences, 1994
- PDGF-AA effectively stimulates early events but has no mitogenic activity in AKR-2B mouse fibroblastsExperimental Cell Research, 1992
- Preparation of biologically active platelet‐derived growth factor isoforms AA and ABEuropean Journal of Biochemistry, 1990
- Preparation of biologically active platelet-derived growth factor type BB from a fusion protein expressed in EscherichiaBiochemistry, 1989