Inhibition of Thrombin Attenuates Stenosis After Arterial Injury in Minipigs

This publication has 30 references indexed in Scilit:

• Reduced acute thrombus formation results in decreased neointimal proliferation after coronary angioplasty
  Journal of the American College of Cardiology, 1995
• The Biologic Behavior of Balloon Hyperinflation–Induced Arterial Lesions in Hypercholesterolemic Pigs Depends on the Presence of Foam Cells
  Arteriosclerosis, Thrombosis, and Vascular Biology, 1995
• Thrombin Generation following Arterial Injury Is a Critical Initiating Event in the Pathogenesis of the Proliferative Stages of the Atherosclerotic Process
  Journal of Vascular Research, 1994
• Cellular and molecular mechanisms of coronary artery restenosis
  Coronary Artery Disease, 1993
• Clot-bound thrombin is protected from inhibition by heparin-antithrombin III but is susceptible to inactivation by antithrombin III-independent inhibitors.
  Journal of Clinical Investigation, 1990
• Tick Anticoagulant Peptide (TAP) Is a Novel Inhibitor of Blood Coagulation Factor Xa
  Science, 1990
• Influence of heparin therapy on percutaneous transluminal coronary angioplasty outcome in patients with coronary arterial thrombus
  The American Journal of Cardiology, 1990
• Effect of 18- to 24-hour heparin administration for prevention of restenosis after uncomplicated coronary angioplasty
  American Heart Journal, 1989
• Monocyte Chemotaxis: Stimulation by Specific Exosite Region in Thrombin
  Science, 1983
• Transluminal coronary angioplasty and early restenosis. Fibrocellular occlusion after wall laceration.
  Heart, 1983