HIV proteins (gp120 and Tat) and methamphetamine in oxidative stress-induced damage in the brain: Potential role of the thiol antioxidant N-acetylcysteine amide
- 24 February 2010
- journal article
- Published by Elsevier in Free Radical Biology & Medicine
- Vol. 48 (10) , 1388-1398
- https://doi.org/10.1016/j.freeradbiomed.2010.02.023
Abstract
No abstract availableKeywords
This publication has 90 references indexed in Scilit:
- Methamphetamine Disrupts Blood–Brain Barrier Function by Induction of Oxidative Stress in Brain Endothelial CellsJournal of Cerebral Blood Flow & Metabolism, 2009
- N-Acetylcysteine amide protects against methamphetamine-induced oxidative stress and neurotoxicity in immortalized human brain endothelial cellsBrain Research, 2009
- Involvement of ROS in BBB dysfunctionFree Radical Research, 2009
- Effects of N-acetylcysteine amide (NACA), a thiol antioxidant on radiation-induced cytotoxicity in Chinese hamster ovary cellsLife Sciences, 2008
- N-acetylcysteine amide (AD4) attenuates oxidative stress in beta-thalassemia blood cellsBiochimica et Biophysica Acta (BBA) - General Subjects, 2008
- Involvement of cytokines in human immunodeficiency virus-1 protein Tat and methamphetamine interactions in the striatumExperimental Neurology, 2006
- Glutathione Pathways in the BrainBiological Chemistry, 2003
- Junctional Adhesion Molecules and Interendothelial JunctionsCells Tissues Organs, 2002
- 4-Hydroxynonenal impairs the permeability of an in vitro rat blood–brain barrierNeuroscience Letters, 2001
- Regional lipid peroxidation and protein oxidation in rat brain after hyperbaric oxygen exposureFree Radical Biology & Medicine, 1996