Intracellular Signaling and Genetic Reprogramming during Agonist-Induced Hypertrophy of Cardiomyocytes
- 1 March 1995
- journal article
- Published by Wiley in Annals of the New York Academy of Sciences
- Vol. 752 (1) , 343-352
- https://doi.org/10.1111/j.1749-6632.1995.tb17443.x
Abstract
No abstract availableThis publication has 57 references indexed in Scilit:
- Angiotensin II receptor antagonist TCV-116 induces regression of hypertensive left ventricular hypertrophy in vivo and inhibits the intracellular signaling pathway of stretch-mediated cardiomyocyte hypertrophy in vitro.Circulation, 1994
- Endothelin ETA receptor antagonist blocks cardiac hypertrophy provoked by hemodynamic overload.Circulation, 1994
- Regulation of hypertrophy and atrophy in cultured adult heart cells.Circulation Research, 1993
- Immediate‐early gene induction by repetitive mechanical but not electrical activity in adult rat cardiomyocytesFEBS Letters, 1993
- Expression of c-fos and Related Genes in the Rat Heart in Response to NorepinephrineJournal of Molecular and Cellular Cardiology, 1993
- Induction of immediate-early genes by angiotensin II and endothelin-1 in adult rat cardiomyocytesJournal Of Hypertension, 1993
- Growth and hypertrophy of the heart: towards an understanding of cardiac specific and inducible gene expressionCardiovascular Research, 1993
- Stimulation of adult rat ventricular myocyte protein synthesis and phosphoinositide hydrolysis by the endothelinsBiochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1993
- Significance of myocardial alpha- and beta-adrenoceptors in catecholamine-induced cardiac hypertrophy.Circulation Research, 1989
- Differentiation of cardiac myocytes after mitogen withdrawal exhibits three sequential states of the ventricular growth response.The Journal of cell biology, 1988