Translationally Controlled Tumor Protein Interacts with the Third Cytoplasmic Domain of Na,K-ATPase α Subunit and Inhibits the Pump Activity in HeLa Cells
Open Access
- 1 November 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (48) , 49868-49875
- https://doi.org/10.1074/jbc.m400895200
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Interaction of Cofilin with Triose-phosphate Isomerase Contributes Glycolytic Fuel for Na,K-ATPase via Rho-mediated Signaling PathwayJournal of Biological Chemistry, 2002
- Intracellular Localization of Na,K-ATPase α2 Subunit MutantsArchives of Biochemistry and Biophysics, 1997
- Ouabain-Induced Cell Proliferation in Cultured Rat AstrocytesThe Japanese Journal of Pharmacology, 1996
- Identification of a Binding Motif for Ankyrin on the α-Subunit of Na+,K+-ATPasePublished by Elsevier ,1995
- GAL11P: A yeast mutation that potentiates the effect of weak GAL4-derived activatorsCell, 1990
- The Na+,K+,2Cl−‐cotransport system in HeLa cells: Aspects of its physiological regulationJournal of Cellular Physiology, 1990
- Structural basis for E1–E2 conformational transitions in Na, K-pump and Ca-pump proteinsThe Journal of Membrane Biology, 1988
- Regulation of rat brain (Na+ + K+)-ATPase activity by cyclic AMPBiochimica et Biophysica Acta (BBA) - Biomembranes, 1982
- Characterization of a new photoaffinity derivative of ouabain: labeling of the large polypeptide and of a proteolipid component of the (sodium-potassium ion)-dependent ATPaseBiochemistry, 1978
- Stimulation of DNA synthesis by ouabain and concanavalin a in cultures of embryonic neural retina cellsCell Differentiation, 1976