Disruption of the GluR2-NSF Interaction Protects Primary Hippocampal Neurons from Ischemic Stress
- 1 April 2001
- journal article
- Published by Elsevier in Molecular and Cellular Neuroscience
- Vol. 17 (4) , 662-670
- https://doi.org/10.1006/mcne.2000.0959
Abstract
No abstract availableKeywords
This publication has 42 references indexed in Scilit:
- Unchanged balance between levels of mRNA encoding AMPA glutamate receptor subtypes following global cerebral ischemia in the rat.Acta Neurologica Scandinavica, 2009
- The AMPA Receptor GluR2 C Terminus Can Mediate a Reversible, ATP-Dependent Interaction with NSF and α- and β-SNAPsNeuron, 1998
- Glutamate receptor subunit 2-selective antibody shows a differential distribution of calcium-impermeable AMPA receptors among populations of neuronsJournal of Comparative Neurology, 1997
- Calcium and Excitotoxic Neuronal InjuryAnnals of the New York Academy of Sciences, 1994
- Changes in gene expression of AMPA-selective glutamate receptor subunits induced by status epilepticus in rat brainNeurochemistry International, 1994
- Growth Factors Prevent Mitochondrial Dysfunction, Loss of Calcium Homeostasis, and Cell Injury, but Not ATP Depletion in Hippocampal Neurons Deprived of GlucoseExperimental Neurology, 1993
- Divalent ion permeability of AMPA receptor channels is dominated by the edited form of a single subunitNeuron, 1992
- NGF and bFGF protect rat hippocampal and human cortical neurons against hypoglycemic damage by stabilizing calcium homeostasisNeuron, 1991
- Blockade of the AMPA receptor prevents CA1 hippocampal injury following severe but transient forebrain ischemia in adult ratsNeuroscience Letters, 1991
- The Role of Glutamate Neurotoxicity in Hypoxic-Ischemic Neuronal DeathAnnual Review of Neuroscience, 1990