Long-term angiotensin-converting enzyme inhibition reduces plasma asymmetric dimethylarginine and improves endothelial nitric oxide bioavailability and coronary microvascular function in patients with syndrome X

This publication has 28 references indexed in Scilit:

• Angiotensin II Stimulates Endothelial Vascular Cell Adhesion Molecule-1 via Nuclear Factor-κB Activation Induced by Intracellular Oxidative Stress
  Arteriosclerosis, Thrombosis, and Vascular Biology, 2000
• Effects of short-term treatment of nicorandil on exercise-induced myocardial ischemia and abnormal cardiac autonomic activity in microvascular angina
  The American Journal of Cardiology, 1997
• Improvement of Coronary Flow Reserve After Long-term Therapy With Enalapril
  Hypertension, 1996
• Elevatedl-Arginine/Dimethylarginine Ratio Contributes to Enhanced Systemic NO Production by Dietaryl-Arginine in Hypercholesterolemic Rabbits
  Biochemical and Biophysical Research Communications, 1996
• Cardiac syndrome X: Clinical characteristics and left ventricular function
  Journal of the American College of Cardiology, 1995
• Effects of angiotensin-converting enzyme inhibition on exercise-induced angina and ST segment depression in patients with microvascular angina
  Journal of the American College of Cardiology, 1994
• Maximal blood flow velocity in severe coronary stenoses measured with a Doppler guidewire: Limitations for the application of the continuity equation in the assessment of stenosis severity
  The American Journal of Cardiology, 1993
• Mechanisms of angina pectoris in syndrome X
  Journal of the American College of Cardiology, 1991
• Effects of enalapril on myocardial noradrenaline overflow during exercise in patients with chronic heart failure.
  Heart, 1989
• Left ventricular function in patients with the anginal syndrome and normal coronary arteriograms
  The American Journal of Cardiology, 1973