Reduced GAP-43 message levels are associated with increased neurofibrillary tangle density in the frontal association cortex (area 9) in Alzheimer's disease
- 31 December 1992
- journal article
- Published by Elsevier in Neurobiology of Aging
- Vol. 13 (6) , 631-639
- https://doi.org/10.1016/0197-4580(92)90085-c
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Neuropathological stageing of Alzheimer-related changesActa Neuropathologica, 1991
- Neurofibrillary degeneration and neuronal loss in alzheimer's diseaseNeurobiology of Aging, 1989
- Localization of the growth-associated phosphoprotein GAP-43 (B-50, F1) in the human cerebral cortexJournal of Neuroscience, 1989
- Decreased levels of protein kinase C in Alzheimer brainBrain Research, 1988
- Neuron numbers and dendritic extent in normal aging and Alzheimer's diseaseNeurobiology of Aging, 1987
- Primary structure and transcriptional regulation of GAP-43, a protein associated with nerve growthCell, 1987
- Quantitative evidence for selective dendritic growth in normal human aging but not in senile dementiaBrain Research, 1981
- Age-related changes in prefrontal cortex of Macaca mulatta: Quantitative analysis of dendritic branching patternsExperimental Neurology, 1980
- Dendritic Growth in the Aged Human Brain and Failure of Growth in Senile DementiaScience, 1979
- Neuronal loss, neurofibrillary tangles and granulovacuolar degeneration in the hippocampus with ageing and dementiaActa Neuropathologica, 1977