PINK1-Associated Parkinson's Disease Is Caused by Neuronal Vulnerability to Calcium-Induced Cell Death
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Open Access
- 1 March 2009
- journal article
- research article
- Published by Elsevier in Molecular Cell
- Vol. 33 (5) , 627-638
- https://doi.org/10.1016/j.molcel.2009.02.013
Abstract
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This publication has 45 references indexed in Scilit:
- Regulation of Mitochondrial Structure and Function by the F1Fo-ATPase Inhibitor Protein, IF1Cell Metabolism, 2008
- PINK1 Is Necessary for Long Term Survival and Mitochondrial Function in Human Dopaminergic NeuronsPLOS ONE, 2008
- Cytoplasmic Pink1 activity protects neurons from dopaminergic neurotoxin MPTPProceedings of the National Academy of Sciences, 2008
- Targeted polyphosphatase expression alters mitochondrial metabolism and inhibits calcium-dependent cell deathProceedings of the National Academy of Sciences, 2007
- Pharmacological inhibition of neuronal NADPH oxidase protects against 1-methyl-4-phenylpyridinium (MPP+)-induced oxidative stress and apoptosis in mesencephalic dopaminergic neuronal cellsNeuroToxicology, 2007
- Impaired dopamine release and synaptic plasticity in the striatum of PINK1 -deficient miceProceedings of the National Academy of Sciences, 2007
- PINK1 Protects against Oxidative Stress by Phosphorylating Mitochondrial Chaperone TRAP1PLoS Biology, 2007
- Mitochondrial pathology and muscle and dopaminergic neuron degeneration caused by inactivation of Drosophila Pink1 is rescued by ParkinProceedings of the National Academy of Sciences, 2006
- Altered cleavage and localization of PINK1 to aggresomes in the presence of proteasomal stressJournal of Neurochemistry, 2006
- The Calcium‐Induced and Sodium‐Induced Effluxes of Calcium from Heart MitochondriaEuropean Journal of Biochemistry, 1977