Current evidence for a modulation of nociception by human genetic polymorphisms
- 1 November 2007
- journal article
- review article
- Published by Wolters Kluwer Health in Pain
- Vol. 132 (1) , 18-22
- https://doi.org/10.1016/j.pain.2007.06.024
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- The search for migraine genes: an overview of current knowledgeCellular and Molecular Life Sciences, 2006
- An SCN9A channelopathy causes congenital inability to experience painNature, 2006
- Catechol- O -methyltransferase gene polymorphisms are associated with multiple pain-evoking stimuliPAIN®, 2006
- Mechanisms of Disease: genetics of fibromyalgiaNature Clinical Practice Rheumatology, 2006
- Mutation in the neuronal voltage-gated sodium channel SCN1A in familial hemiplegic migraineThe Lancet, 2005
- Genetic basis for individual variations in pain perception and the development of a chronic pain conditionHuman Molecular Genetics, 2004
- Effect of the A118G polymorphism on binding affinity, potency and agonist‐mediated endocytosis, desensitization, and resensitization of the human mu‐opioid receptorJournal of Neurochemistry, 2004
- Opioid receptor random mutagenesis reveals a mechanism for G protein–coupled receptor activationNature Structural & Molecular Biology, 2003
- Familial Dysautonomia Is Caused by Mutations of the IKAP GeneAmerican Journal of Human Genetics, 2001
- Single-nucleotide polymorphism in the human mu opioid receptor gene alters β-endorphin binding and activity: Possible implications for opiate addictionProceedings of the National Academy of Sciences, 1998