Regulation of Adipogenesis by a Transcriptional Repressor That Modulates MAPK Activation
Open Access
- 1 March 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (13) , 10199-10206
- https://doi.org/10.1074/jbc.m010640200
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Inhibition of Adipogenesis Through MAP Kinase-Mediated Phosphorylation of PPARγScience, 1996
- A Novel Cytoplasmic Dual Specificity Protein Tyrosine Phosphatase Implicated in Muscle and Neuronal DifferentiationJournal of Biological Chemistry, 1996
- A eukaryotic transcriptional represser with carboxypeptidase activityNature, 1995
- Inactivation of p42 MAP kinase by protein phosphatase 2A and a protein tyrosine phosphatase, but not CL100, in various cell linesCurrent Biology, 1995
- Epitope-tag vectors for eukaryotic protein productionGene, 1995
- Stimulation of adipogenesis in fibroblasts by PPARγ2, a lipid-activated transcription factorCell, 1994
- Transformation of Mammalian Cells by Constitutively Active MAP Kinase KinaseScience, 1994
- Ectopic expression of the CCAAT/enhancer-binding protein alpha promotes the adipogenic program in a variety of mouse fibroblastic cells.Genes & Development, 1994
- mPPAR gamma 2: tissue-specific regulator of an adipocyte enhancer.Genes & Development, 1994
- Differential effects of fibroblast growth factor and tumor promoters on the initiation and maintenance of adipocyte differentiation.The Journal of cell biology, 1989