Expression of MK6a dominant-negative and C-terminal mutant transgenes in mice has distinct phenotypic consequences in the epidermis and hair follicle
- 1 October 1999
- journal article
- research article
- Published by Elsevier in Differentiation
- Vol. 65 (2) , 97-112
- https://doi.org/10.1046/j.1432-0436.1999.6520097.x
Abstract
No abstract availableKeywords
This publication has 60 references indexed in Scilit:
- A Novel Human Type II Cytokeratin, K6hf, Specifically Expressed in the Companion Layer of the Hair FollicleJournal of Investigative Dermatology, 1998
- Missense Mutations in Keratin 17 Cause Either Pachyonychia Congenita Type 2 or a Phenotype Resembling Steatocystoma MultiplexJournal of Investigative Dermatology, 1997
- Human keratin diseases:Experimental Dermatology, 1996
- Regulation of Epidermal Expression of Keratin K17 in Inflammatory Skin DiseasesJournal of Investigative Dermatology, 1996
- 14-3-3 proteins associate with phosphorylated simple epithelial keratins during cell cycle progression and act as a solubility cofactor.The Journal of cell biology, 1996
- Epidermal disease: faulty keratin filaments take their tollNature Genetics, 1994
- Genetic skin diseases caused by mutations in keratin intermediate filamentsTrends in Genetics, 1993
- Aberrant expression during two‐stage mouse skin carcinogenesis of a type 147‐kDa keratin, k13, normally associated with terminal differentiation of internal stratified epitheliaMolecular Carcinogenesis, 1988
- Development and Morphology of the Periderm of Mouse Embryos (Days 9–12 of Gestation)Cells Tissues Organs, 1988
- Electron Microscopic Study on Cell Differentiation in Anagen Hair Follicles in MiceJournal of Investigative Dermatology, 1988