Observations on Steatorrhea Associated with Gastric Hypersecretion and Pancreatic Islet Cell Neoplasm

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Abstract
A case illustrating a pancreatic islet cell neoplasm associated with gastric hypersecretion and steatorrhea is described. During successive test periods, the fecal loss of fat was eliminated when all acid gastric secretions were aspirated, and was practically normal when all gastric aspirates were neutralized and returned to the duodenum. A year of remission in diarrhea followed a subtotal gastrectomy. It is postulated that the steatorrhea in this syndrome derives from an inactivation of essential digestive enzyme systems in the lumen of the small intestine.

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