Structural Determinants of KvLQT1 Control by the KCNE Family of Proteins

This publication has 25 references indexed in Scilit:

• A constitutively open potassium channel formed by KCNQ1 and KCNE3
  Nature, 2000
• A superfamily of small potassium channel subunits: form and function of the MinK-related peptides (MiRPs)
  Quarterly Reviews of Biophysics, 1998
• Voltage‐dependent inactivation of the human K⁺ channel KvLQT1 is eliminated by association with minimal K⁺ channel (minK) subunits
  The Journal of Physiology, 1998
• Mutation of the Gene for I sK Associated With Both Jervell and Lange-Nielsen and Romano-Ward Forms of Long-QT Syndrome
  Circulation, 1998
• IsK and KvLQT1: Mutation in Either of the Two Subunits of the Slow Component of the Delayed Rectifier Potassium Channel Can Cause Jervell and Lange-Nielsen Syndrome
  Human Molecular Genetics, 1997
• KvLQT1, a voltage-gated potassium channel responsible for human cardiac arrhythmias
  Proceedings of the National Academy of Sciences, 1997
• A novel mutation in the potassium channel gene KVLQT1 causes the Jervell and Lange-Nielsen cardioauditory syndrome
  Nature Genetics, 1997
• Coassembly of KVLQT1 and minK (IsK) proteins to form cardiac IKS potassium channel
  Nature, 1996
• KvLQT1 and IsK (minK) proteins associate to form the IKS cardiac potassium current
  Nature, 1996
• Positional cloning of a novel potassium channel gene: KVLQT1 mutations cause cardiac arrhythmias
  Nature Genetics, 1996