Serum Enzyme Activities Following Morphine

Abstract
An increase of serum glutamic oxalacetic transaminase (SGO-T) activity following myocardial infarction was first described by LaDue et al.1and subsequently confirmed by numerous investigators.2-6Much emphasis has been placed on elevated SGO-T values in substantiating the clinical impression of myocardial infarction, especially when the electrocardiogram reveals equivocal changes, previous infarction, left bundle branch block, or arrhythmia.7-9With increased understanding of the ubiquitous nature of GO-T distribution in human tissues, it has become apparent that liver necrosis10-14and a host of other disease states5,6,15-21can provoke a rise in SGO-T activity. Thus, confusion may still exist when there is elevated SGO-T activity in the absence of electrocardiographic evidence of heart muscle damage. Obstructive biliary tract disease causes a rise in SGO-T activity,22,23and this rise may be unrelated to hepatic necrosis.24The demonstration of high GO-T activity in bile25further serves to

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