Loss of CD23 Is a Consequence of B‐Cell Activation
- 1 May 1992
- journal article
- Published by Wiley in Annals of the New York Academy of Sciences
- Vol. 651 (1) , 130-142
- https://doi.org/10.1111/j.1749-6632.1992.tb24602.x
Abstract
Click on the article title to read more.Keywords
This publication has 32 references indexed in Scilit:
- Treatment of murine CD5− B cells with anti-lg, but not LPS, induces surface CD5: two B-cell activation pathwaysInternational Immunology, 1991
- Genetic basis of the neonatal antibody repertoire: germline V-gene expression and limited N-region diversityInternational Immunology, 1990
- FcεRII/CD23: The Low Affinity Receptor for IgEAnnual Review of Immunology, 1990
- Normal, Autoimmune, and Malignant CD5+ B Cells: The LY-1 B Lineage?Annual Review of Immunology, 1988
- Human recombinant interleukin 4 induces Fc epsilon receptors (CD23) on normal human B lymphocytes.The Journal of Experimental Medicine, 1987
- Role of membrane immunoglobulin (Ig) crosslinking in membrane Ig-mediated, major histocompatibility-restricted T cell-B cell cooperation.The Journal of Experimental Medicine, 1985
- Preferential utilization of the most JH-proximal VH gene segments in pre-B-cell linesNature, 1984
- The "Ly-1 B" cell subpopulation in normal immunodefective, and autoimmune mice.The Journal of Experimental Medicine, 1983
- Precommitment of normal mouse peritoneal cells by erythrocyte antigens in relation to auto-antibody productionNature, 1975
- Induction of B cell tolerance to polysaccharides by exhaustive immunization and during immunosuppression with cyclophosphamideEuropean Journal of Immunology, 1974