The ubiquitously expressed MURR1 protein is absent in canine copper toxicosis
- 1 November 2003
- journal article
- Published by Elsevier in Journal of Hepatology
- Vol. 39 (5) , 703-709
- https://doi.org/10.1016/s0168-8278(03)00380-5
Abstract
No abstract availableKeywords
This publication has 23 references indexed in Scilit:
- Wilson disease and Menkes disease: new handles on heavy-metal transportPublished by Elsevier ,2002
- Biochemical Characterization of the Human Copper Transporter Ctr1Journal of Biological Chemistry, 2002
- Diagnostic value of a microsatellite DNA marker for copper toxicosis in West-European Bedlington terriers and incidence of the diseaseAnimal Genetics, 1999
- Association of copper to metallothionein in hepatic lysosomes of Long–Evans cinnamon (LEC) rats during the development of hepatitisEuropean Journal of Clinical Investigation, 1998
- Functional Expression of the Menkes Disease Protein Reveals Common Biochemical Mechanisms Among the Copper-transporting P-type ATPasesJournal of Biological Chemistry, 1998
- Human Short-Chain L-3-Hydroxyacyl-CoA Dehydrogenase: Cloning and Characterization of the Coding SequenceBiochemical and Biophysical Research Communications, 1996
- The Wilson disease gene is a copper transporting ATPase with homology to the Menkes disease geneNature Genetics, 1993
- The Wilson disease gene is a putative copper transporting P–type ATPase similar to the Menkes geneNature Genetics, 1993
- Isolation and Characterization of a Human Liver cDNA as a Candidate Gene for Wilson DiseaseBiochemical and Biophysical Research Communications, 1993
- Biliary copper excretion by hepatocyte lysosomes in the rat. Major excretory pathway in experimental copper overload.Journal of Clinical Investigation, 1989