Protein tyrosine kinase inhibitor, genistein, enhances apoptosis and cell cycle arrest in K562 cells treated with γ-irradiation
- 5 October 2000
- journal article
- research article
- Published by Elsevier in Cancer Letters
- Vol. 160 (1) , 107-113
- https://doi.org/10.1016/s0304-3835(00)00569-3
Abstract
No abstract availableKeywords
This publication has 16 references indexed in Scilit:
- Protein Kinase Cι Activity Is Necessary for Bcr-Abl-mediated Resistance to Drug-induced ApoptosisJournal of Biological Chemistry, 1999
- Herbimycin A accelerates the induction of apoptosis following etoposide treatment or γ-irradiation of bcr/abl-positive leukaemia cellsOncogene, 1998
- Bcl-2-independent Bcr–Abl-mediated resistance to apoptosis: protection is correlated with up regulation of Bcl-xLOncogene, 1998
- Cell-cycle arrest versus cell death in cancer therapyNature Medicine, 1997
- Down-Modulation of P210bcr/abl Induces Apoptosis/Differentiation in K562 Leukemic Blast CellsTumori Journal, 1997
- Cell Cycle Checkpoints: Preventing an Identity CrisisScience, 1996
- UCN-01: a Potent Abrogator of G2 Checkpoint Function in Cancer Cells With Disrupted p53JNCI Journal of the National Cancer Institute, 1996
- Regulation of the Fas Apoptotic Cell Death Pathway by AblPublished by Elsevier ,1995
- Properties of the K562 cell line, derived from a patient with chronic myeloid leukemiaInternational Journal of Cancer, 1976
- Human chronic myelogenous leukemia cell-line with positive Philadelphia chromosomeBlood, 1975