Tolerance and Sensitization to Endotoxin in Kupffer Cells Caused by Acute Ethanol Involve Interleukin-1 Receptor-Associated Kinase
- 2 November 2000
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 277 (3) , 686-690
- https://doi.org/10.1006/bbrc.2000.3738
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- Essential role of tumor necrosis factor α in alcohol-induced liver injury in miceGastroenterology, 1999
- Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 GeneScience, 1998
- Antibodies to tumor necrosis factor alfa attenuate hepatic necrosis and inflammation caused by chronic exposure to ethanol in the ratHepatology, 1997
- MyD88: An Adapter That Recruits IRAK to the IL-1 Receptor ComplexImmunity, 1997
- Cross‐Tolerance Between Acute Alcohol Intoxication and EndotoxemiaAlcohol, Clinical and Experimental Research, 1996
- Antibiotics prevent liver injury in rats following long-term exposure to ethanolGastroenterology, 1995
- Inactivation of Kupffer cells prevents early alcohol-induced liver injuryHepatology, 1994
- Alcohol Administration Attenuates LPS-Induced Expression of Inducible Nitric Oxide Synthase in Kupffer and Hepatic Endothelial CellsBiochemical and Biophysical Research Communications, 1993
- Dominant and recessive mutations define functional domains of Toll, a transmembrane protein required for dorsal-ventral polarity in the Drosophila embryo.Genes & Development, 1991
- Acute Alcohol Infusion Suppresses Endotoxin‐induced Serum Tumor Necrosis FactorAlcohol, Clinical and Experimental Research, 1989