Cardiotoxicity of Naja nigricollis phospholipase A2 is not due to alterations in prostaglandin synthesis
- 1 January 1986
- Vol. 24 (11-12) , 1107-1116
- https://doi.org/10.1016/0041-0101(86)90137-6
Abstract
No abstract availableThis publication has 40 references indexed in Scilit:
- Cardiotoxic effects of venom phospholipase A2 are not due to phospholipid hydrolytic productsLife Sciences, 1984
- Effect of carboxylate group modification on enzymatic and cardiotoxic properties of snake venom phospholipases A2Toxicon, 1984
- Prostanoids can modify response to electrical stimulus and 45Ca2+ exchange in isolated myocardial muscle cellsProstaglandins, Leukotrienes and Medicine, 1983
- Developmental alterations in 5′-nucleotidase kinetics and lipid composition of rat heart sarcolemmaMechanisms of Ageing and Development, 1983
- Ethoxyformylation and guanidination of snake venom phospholipases A2: Effects on enzymatic activity, lethality and some pharmacological propertiesToxicon, 1983
- Dissociation of enzymatic activity from lethality and pharmacological properties by carbamylation of lysines in Naja nigricollis and Naja naja atra snake venom phospholipases A2Toxicon, 1981
- Prostaglandin synthesis by adult heart myocytesJournal of Molecular and Cellular Cardiology, 1980
- Synthesis of prostaglandins by cultured rat heart myocytes and cardiac mesenchymal cellsJournal of Molecular and Cellular Cardiology, 1980
- Comparison of a relatively toxic phospholipase A2 from Naja nigricollis snake venom with that of a relatively non-toxic phospholipase A2 from Hemachatus haemachatus snake venom—IBiochemical Pharmacology, 1980
- Demonstration of phospholipid splitting as the factor responsible for increased permeability and block of axonal conduction induced by snake venom. I. Study on lobster axonsBiochimica et Biophysica Acta (BBA) - Biomembranes, 1967