Epstein-Barr Virus Lytic Replication Elicits ATM Checkpoint Signal Transduction While Providing an S-phase-like Cellular Environment

This publication has 50 references indexed in Scilit:

• Inhibition of S-Phase Cyclin-Dependent Kinase Activity Blocks Expression of Epstein-Barr Virus Immediate-Early and Early Genes, Preventing Viral Lytic Replication
  Journal of Virology, 2004
• The EBNA- 3 gene family proteins disrupt the G2/M checkpoint
  Oncogene, 2003
• Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaks
  Nature Cell Biology, 2003
• DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociation
  Nature, 2003
• Reactivation of Lytic Replication from B Cells Latently Infected with Epstein-Barr Virus Occurs with High S-Phase Cyclin-Dependent Kinase Activity while Inhibiting Cellular DNA Replication
  Journal of Virology, 2003
• The Epstein-Barr Virus Immediate-Early Protein BZLF1 Induces Expression of E2F-1 and Other Proteins Involved in Cell Cycle Progression in Primary Keratinocytes and Gastric Carcinoma Cells
  Journal of Virology, 2002
• A Murine Model of Nijmegen Breakage Syndrome
  Current Biology, 2002
• The DNA damage-dependent intra–S phase checkpoint is regulated by parallel pathways
  Nature Genetics, 2002
• WAF1, a potential mediator of p53 tumor suppression
  Cell, 1993
• Identification and characterization of oriLyt, a lytic origin of DNA replication of Epstein-Barr virus
  Cell, 1988