Chelatable cellular copper modulates differentiation and self-renewal of cord blood–derived hematopoietic progenitor cells
- 1 October 2005
- journal article
- Published by Elsevier in Experimental Hematology
- Vol. 33 (10) , 1092-1100
- https://doi.org/10.1016/j.exphem.2005.06.015
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- NGF promotes copper accumulation required for optimum neurite outgrowth and protein methylationJournal of Neurobiology, 2005
- Copper induces histone hypoacetylation through directly inhibiting histone acetyltransferase activityChemico-Biological Interactions, 2004
- Linear polyamine copper chelator tetraethylenepentamine augments long-term ex vivo expansion of cord blood-derived CD34+ cells and increases their engraftment potential in NOD/SCID miceExperimental Hematology, 2004
- -Catenin contributes to leukemogenesis induced by AML-associated translocation products by increasing the self-renewal of very primitive progenitor cellsBlood, 2004
- Design, synthesis and properties of novel iron(III)-specific fluorescent probesJournal of Pharmacy and Pharmacology, 2004
- Murine Bone Marrow Cells Cultured Ex Vivo in the Presence of Multiple Cytokine Combinations Lose Radioprotective and Long-Term Engraftment PotentialStem Cells and Development, 2004
- Cellular copper content modulates differentiation and self‐renewal in cultures of cord blood‐derived CD34+ cellsBritish Journal of Haematology, 2002
- Role for Copper in Transient Oxidation and Nuclear Translocation of MTF-1, but Not of NF-κB, by the Heme–Hemopexin Transport SystemAntioxidants and Redox Signaling, 2000
- REDOX REGULATION OF CELLULAR ACTIVATIONAnnual Review of Immunology, 1997
- The effect of tetrathiomolybdate on the metabolism of copper by hepatocytes and fibroblastsBiological Trace Element Research, 1989