Monocyte/macrophage response to β2-microglobulin modified with advanced glycation end products
- 1 February 1996
- journal article
- Published by Elsevier in Kidney International
- Vol. 49 (2) , 538-550
- https://doi.org/10.1038/ki.1996.77
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Is β2-microglobulin a mediator of bone disease?Kidney International, 1995
- Glycation of Human .beta.2-Microglobulin in Patients with Hemodialysis-Associated Amyloidosis: Identification of the Glycated SitesBiochemistry, 1994
- beta 2-Microglobulin modified with advanced glycation end products is a major component of hemodialysis-associated amyloidosis.Journal of Clinical Investigation, 1993
- Effect of dialysis membrane and patient's age on signs of dialysis-related amyloidosisKidney International, 1991
- Role of Oxidative Stress in Development of Complications in DiabetesDiabetes, 1991
- Advanced protein glycosylation induces transendothelial human monocyte chemotaxis and secretion of platelet-derived growth factor: role in vascular disease of diabetes and aging.Proceedings of the National Academy of Sciences, 1990
- Cachectin/TNF and IL-1 Induced by Glucose-Modified Proteins: Role in Normal Tissue RemodelingScience, 1988
- Advanced Glycosylation End Products in Tissue and the Biochemical Basis of Diabetic ComplicationsNew England Journal of Medicine, 1988
- Polymerization of intact beta 2-microglobulin in tissue causes amyloidosis in patients on chronic hemodialysis.Proceedings of the National Academy of Sciences, 1986
- A new form of amyloid protein associated with chronic hemodialysis was identified as β2-microglobulinBiochemical and Biophysical Research Communications, 1985