Repeated amphetamine treatment causes a persistent elevation of glial fibrillary acidic protein in the caudate–putamen
- 19 March 2004
- journal article
- research article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 488 (1-3) , 111-115
- https://doi.org/10.1016/j.ejphar.2004.02.001
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- Repeated Administration of Amphetamine or Cocaine Does Not Alter AMPA Receptor Subunit Expression in the Rat Midbrain,Neuropsychopharmacology, 2002
- The expression of glutamate transporter GLT-1 in the rat cerebral cortex is down-regulated by the antipsychotic drug clozapineMolecular Psychiatry, 2001
- Effects of methamphetamine-induced neurotoxicity on the development of neural circuitry: a hypothesisBrain Research Reviews, 2000
- Review Article The family of sodium-dependent glutamate transporters: a focus on the GLT-1/EAAT2 subtypeNeurochemistry International, 1998
- GLIA-NEURON INTERCOMMUNICATIONS AND SYNAPTIC PLASTICITYProgress in Neurobiology, 1996
- The effect of repeated amphetamine treatment on striatal DA transporter and rotation in ratsBrain Research, 1994
- Dopamine transmission in the initiation and expression of drug- and stress-induced sensitization of motor activityBrain Research Reviews, 1991
- Suppression by antisense mRNA demonstrates a requirement for the glial fibrillary acidic protein in the formation of stable astrocytic processes in response to neurons.The Journal of cell biology, 1991
- Lack of increase in dopamine transporter binding or function in rat brain tissue after treatment with blockers of neuronal uptake of dopamineNeuropharmacology, 1991
- Fluoxetine increases long-lasting neostriatal dopamine depletion after administration of d-methamphetamine and d-amphetamineNeuropharmacology, 1983