An HSV-TK Transgenic Mouse Model to Evaluate Elimination of Fibroblasts for Fibrosis Therapy
- 31 August 2003
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 163 (2) , 789-801
- https://doi.org/10.1016/s0002-9440(10)63706-6
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- β1 Integrin Regulates Fibroblast Viability during Collagen Matrix Contraction through a Phosphatidylinositol 3-Kinase/Akt/Protein Kinase B Signaling PathwayJournal of Biological Chemistry, 2002
- Localization of Changes inβ-actin Expression in Remodeled Canine MyocardiumJournal of Molecular and Cellular Cardiology, 1999
- Progressive Renal Disease: Fibroblasts, Extracellular Matrix, and IntegrinsNephron Experimental Nephrology, 1999
- A potent far-upstream enhancer in the mouse pro alpha 2(I) collagen gene regulates expression of reporter genes in transgenic mice.The Journal of cell biology, 1996
- CD44-related chondroitin sulfate proteoglycan, a cell surface receptor implicated with tumor cell invasion, mediates endothelial cell migration on fibrinogen and invasion into a fibrin matrix.Journal of Clinical Investigation, 1996
- Keratinocyte growth factor and hepatocyte growth factor/scatter factor are heparin-binding growth factors for alveolar type II cells in fibroblast-conditioned medium.Journal of Clinical Investigation, 1993
- Mesenchymal cells isolated after acute lung injury manifest an enhanced proliferative phenotype.Journal of Clinical Investigation, 1992
- Mechanisms and Functions of Cell DeathAnnual Review of Cell Biology, 1991
- Identification and partial characterization of angiogenesis bioactivity in the lower respiratory tract after acute lung injury.Journal of Clinical Investigation, 1991
- Transgenic mice with inducible dwarfismNature, 1989