Could low-level background exposure to persistent organic pollutants contribute to the social burden of type 2 diabetes?

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Abstract
Persistent organic pollutants (POPs) include hundreds of different chemical compounds with common properties, such as long-term persistence, widespread diffusion in the environment and bioaccumulation through the food chain.1–3 In various species, POPs are linked to cancer, neurobehavioural disorders, impaired immunity, endocrine problems and reproductive disorders. Most epidemiological findings to date have focused on people with high exposure to POPs in occupational or accidental settings, whereas people without such high exposure have been much less studied; with few exceptions, this approach has uncovered only modest associations with various health outcomes.2,4