Inducible IL-23p19 expression in human microglia via p38 MAPK and NF-κB signal pathways

This publication has 37 references indexed in Scilit:

• p38 Mitogen-Activated Protein Kinase Controls NF-κB Transcriptional Activation and Tumor Necrosis Factor Alpha Production through RelA Phosphorylation Mediated by Mitogen- and Stress-Activated Protein Kinase 1 in Response toBorrelia burgdorferiAntigens
  Infection and Immunity, 2007
• Anti-IL-23 therapy inhibits multiple inflammatory pathways and ameliorates autoimmune encephalomyelitis
  Journal of Clinical Investigation, 2006
• Transforming growth factor-β induces development of the TH17 lineage
  Nature, 2006
• Intracellular network of phosphatidylinositol 3-kinase, mammalian target of the rapamycin/70 kDa ribosomal S6 kinase 1, and mitogen-activated protein kinases pathways for regulating mycobacteria-induced IL-23 expression in human macrophages
  Cellular Microbiology, 2006
• A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17
  Nature Immunology, 2005
• IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
  The Journal of Experimental Medicine, 2005
• LPS, TLR4 and infectious disease diversity
  Nature Reviews Microbiology, 2005
• Attenuation of Murine Collagen-Induced Arthritis by a Novel, Potent, Selective Small Molecule Inhibitor of IκB Kinase 2, TPCA-1 (2-[(Aminocarbonyl)amino]-5-(4-fluorophenyl)-3-thiophenecarboxamide), Occurs via Reduction of Proinflammatory Cytokines and Antigen-Induced T Cell Proliferation
  The Journal of Pharmacology and Experimental Therapeutics, 2005
• The involvement of AU-rich element-binding proteins in p38 mitogen-activated protein kinase pathway-mediated mRNA stabilisation
  Cellular Signalling, 2004
• SB 203580 is a specific inhibitor of a MAP kinase homologue which is stimulated by cellular stresses and interleukin‐1
  Published by Wiley ,2000