Components of hypothalamic obesity: bipiperidyl-mustard lesions add hyperphagia to monosodium glutamate-induced hyperinsulinemia
- 1 May 1986
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 374 (2) , 380-384
- https://doi.org/10.1016/0006-8993(86)90434-8
Abstract
No abstract availableKeywords
This publication has 15 references indexed in Scilit:
- Bipiperidyl mustard produces brain lesions and obesity in the ratBrain Research, 1981
- The role of hyperinsulinema and the vagus nerve in hypothalamic hyperphagia reexaminedDiabetologia, 1981
- Neural pathways involved in the hypothalamic integration of autonomic responsesDiabetologia, 1981
- Cellular basis of direct insulin action in the central nervous systemDiabetologia, 1981
- The induction of obesity in the rat with bipiperidyl mustardBritish Journal of Nutrition, 1980
- Effects of hyothalamic lesions at different loci on development of hyperinsulinemia and obesity in the weanling ratJournal of Comparative Neurology, 1971
- Brain Lesions, Obesity, and Other Disturbances in Mice Treated with Monosodium GlutamateScience, 1969
- Obesity and EatingScience, 1968
- Bipiperidyl Mustard, A New Obesifying Agent in the MouseScience, 1966
- Hypothalamic lesions and adiposity in the ratThe Anatomical Record, 1940