Premature senescence of mesothelial cells is associated with non-telomeric DNA damage
- 20 August 2007
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 362 (3) , 707-711
- https://doi.org/10.1016/j.bbrc.2007.08.047
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- Early loss of proliferative potential of human peritoneal mesothelial cells in culture: the role of p16INK4a-mediated premature senescenceJournal of Applied Physiology, 2006
- Automatic and Quantitative Measurement of Protein-Protein Colocalization in Live CellsBiophysical Journal, 2004
- Cancer and ageing: rival demons?Nature Reviews Cancer, 2003
- Erosion of the telomeric single-strand overhang at replicative senescenceNature Genetics, 2003
- Peritoneal Dialysis and Epithelial-to-Mesenchymal Transition of Mesothelial CellsNew England Journal of Medicine, 2003
- Mesothelial cells: Their structure, function and role in serosal repairRespirology, 2002
- Putative telomere-independent mechanisms of replicative aging reflect inadequate growth conditionsGenes & Development, 2001
- Accumulation of single-strand breaks is the major cause of telomere shortening in human fibroblastsFree Radical Biology & Medicine, 2000
- Both Rb/p16INK4a inactivation and telomerase activity are required to immortalize human epithelial cellsNature, 1998
- Telomeres shorten during ageing of human fibroblastsNature, 1990