Molecular characterization of a complex chromosomal rearrangement in a pleomorphic salivary gland adenoma involving the 3′-UTR of HMGIC

This publication has 21 references indexed in Scilit:

• LPP,the Preferred Fusion Partner Gene ofHMGICin Lipomas, Is a Novel Member of the LIM Protein Gene Family
  Genomics, 1996
• Cytogenetics and Molecular Genetics of Bone and Soft Tissue Tumors
  Advances in Cancer Research, 1996
• A 6-Mb Yeast Artificial Chromosome Contig and Long-Range Physical Map Encompassing the Region on Chromosome 12q15 Frequently Rearranged in a Variety of Benign Solid Tumors
  Genomics, 1995
• Recurrent rearrangements in the high mobility group protein gene, HMGI-C, in benign mesenchymal tumours
  Nature Genetics, 1995
• Disruption of the architectural factor HMGI-C: DNA-binding AT hook motifs fused in lipomas to distinct transcriptional regulatory domains
  Cell, 1995
• Molecular characterization of mar, a multiple aberration region on human chromosome segment 12q13-q15 implicated in various solid tumors
  Genes, Chromosomes and Cancer, 1995
• Physical Mapping of Chromosome 12q Breakpoints in Lipoma, Pleomorphic Salivary Gland Adenoma, Uterine Leiomyoma, and Myxoid Liposarcoma
  Genomics, 1994
• Cytogenetic subtyping of 220 salivary gland pleomorphic adenomas: Correlation to occurrence, histological subtype, and in vitro cellular behavior
  Cancer Genetics and Cytogenetics, 1993
• Isolation of a somatic cell hybrid retaining the der(16)t(12;16)(q13;p11.2) from a myxoid liposarcoma cell line
  Cytogenetic and Genome Research, 1993
• Cytogenetic and molecular observations in human and experimental salivary gland tumors
  Cancer Genetics and Cytogenetics, 1990