Bcl-xL Acts Downstream of Caspase-8 Activation by the CD95 Death-inducing Signaling Complex
Open Access
- 1 February 1998
- journal article
- Published by Elsevier
- Vol. 273 (6) , 3388-3393
- https://doi.org/10.1074/jbc.273.6.3388
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Resistance of cultured peripheral T cells towards activation‐induced cell death involves a lack of recruitment of FLICE (MACH/caspase 8) to the CD95 death‐inducing signaling complexEuropean Journal of Immunology, 1997
- Interaction between the C. elegans cell-death regulators CED-9 and CED-4Nature, 1997
- Apoptosis: Telling cells their time is upCurrent Biology, 1996
- FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling ComplexCell, 1996
- Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell DeathCell, 1996
- ICE Family Proteases: Mediators of All Apoptotic Cell Death?Immunity, 1996
- Molecular Ordering of the Cell Death Pathway: Bcl-2 AND Bcl-xL FUNCTION UPSTREAM OF THE CED-3-LIKE APOPTOTIC PROTEASESPublished by Elsevier ,1996
- Developing Caenorhabditis elegans neurons may contain both cell-death protective and killer activities.Genes & Development, 1996
- The C. elegans cell death gene ced-3 encodes a protein similar to mammalian interleukin-1β-converting enzymeCell, 1993
- Mechanisms and Functions of Cell DeathAnnual Review of Cell Biology, 1991