Dominant Negative Farnesyltransferase α-Subunit Inhibits Insulin Mitogenic Effects

This publication has 18 references indexed in Scilit:

• Insulin Signals to Prenyltransferases via the Shc Branch of Intracellular Signaling
  Published by Elsevier ,2001
• Insulin Potentiates Platelet-Derived Growth Factor Action in Vascular Smooth Muscle Cells*This work was supported by grants from the VA Research Service, the NIH (HL-48920 to R.N., HD-24497–06 to J.S.), PO-1-HL-55798 (to J.N. and R.N.), jointly funded by the NIH and the Juvenile Diabetes Foundation, the American Heart Association, a fellowship support from the American Heart Association (to M.G.), HealthONE Foundation: Diabetes and Endocrine Research and Education Fund, and Foundation for Biomedical Education and Research.
  Endocrinology, 1998
• Effect of Insulin on Farnesyltransferase
  Journal of Biological Chemistry, 1998
• Insulin Stimulates the Phosphorylation and Activity of Farnesyltransferase via the Ras-Mitogen-Activated Protein Kinase Pathway*
  Endocrinology, 1997
• Effect of Insulin on Farnesyltransferase Activity in 3T3-L1 Adipocytes
  Journal of Biological Chemistry, 1996
• PROTEIN PRENYLATION: Molecular Mechanisms and Functional Consequences
  Annual Review of Biochemistry, 1996
• Characterization of recombinant human farnesyl-protein transferase: Cloning, expression, farnesyl diphosphate binding, and functional homology with yeast prenyl-protein transferases
  Biochemistry, 1993
• Identification and preliminary characterization of protein-cysteine farnesyltransferase.
  Proceedings of the National Academy of Sciences, 1990
• p21ras is modified by a farnesyl isoprenoid.
  Proceedings of the National Academy of Sciences, 1989
• The p21 ras C-terminus is required for transformation and membrane association
  Nature, 1984