PU.1, Interferon Regulatory Factor (IRF) 2, and the Interferon Consensus Sequence-binding Protein (ICSBP/IRF8) Cooperate to Activate NF1 Transcription in Differentiating Myeloid Cells

This publication has 41 references indexed in Scilit:

• Leukemia-Associated, Constitutively Active Mutants of SHP2 Protein Tyrosine Phosphatase Inhibit NF1 Transcriptional Activation by the Interferon Consensus Sequence Binding Protein
  Molecular and Cellular Biology, 2006
• Activating Mutations of the Noonan Syndrome-Associated SHP2/PTPN11 Gene in Human Solid Tumors and Adult Acute Myelogenous Leukemia
  Cancer Research, 2004
• Nramp1-mediated Innate Resistance to Intraphagosomal Pathogens Is Regulated by IRF-8, PU.1, and Miz-1
  Published by Elsevier ,2003
• The prognostic significance of bone marrow levels of neurofibromatosis‐1 protein and ras oncogene mutations in patients with acute myeloid leukemia and myelodysplastic syndrome
  Cancer, 2003
• Quantitative Hydroxyl Radical Footprinting Reveals Cooperative Interactions between DNA-Binding Subdomains of PU.1 and IRF4
  Biochemistry, 1998
• Interferon Regulatory Factor-2 Directs Transcription from the gp91 Promoter
  Published by Elsevier ,1996
• Loss of NF1 results in activation of the Ras signaling pathway and leads to aberrant growth in haematopoietic cells
  Nature Genetics, 1996
• Nf1 deficiency causes Ras-Dediated granulocyte/macrophage colony stimulating factor hypersensitivity and chronic myeloid leukaemia
  Nature Genetics, 1996
• Requirement of Transcription Factor PU.1 in the Development of Multiple Hematopoietic Lineages
  Science, 1994
• Aberrant regulation of ras proteins in malignant tumour cells from type 1 neurofibromatosis patients
  Nature, 1992