Overexpression of Helicard, a CARD-Containing Helicase Cleaved during Apoptosis, Accelerates DNA Degradation
- 1 May 2002
- journal article
- research article
- Published by Elsevier in Current Biology
- Vol. 12 (10) , 838-843
- https://doi.org/10.1016/s0960-9822(02)00842-4
Abstract
No abstract availableKeywords
Funding Information
- Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung
- Association pour la Recherche sur le Cancer
- Australian Research Council
This publication has 21 references indexed in Scilit:
- Mitochondrial endonuclease G is important for apoptosis in C. elegansNature, 2001
- Endonuclease G is an apoptotic DNase when released from mitochondriaNature, 2001
- Mechanisms for ATP-dependent chromatin remodellingCurrent Opinion in Genetics & Development, 2001
- Apoptotic DNA FragmentationExperimental Cell Research, 2000
- Acinus is a caspase-3-activated protein required for apoptotic chromatin condensationNature, 1999
- The modular nature of apoptotic signaling proteinsCellular and Molecular Life Sciences, 1999
- Molecular characterization of mitochondrial apoptosis-inducing factorNature, 1999
- Unraveling the role of helicases in transcriptionBioEssays, 1998
- Cleavage of CAD inhibitor in CAD activation and DNA degradation during apoptosisNature, 1998
- DNA helicases in inherited human disordersCurrent Opinion in Genetics & Development, 1997