Why Does Airway Inflammation Persist? Is It Leukotrienes?
- 1 March 2000
- journal article
- review article
- Published by American Thoracic Society in American Journal of Respiratory and Critical Care Medicine
- Vol. 161 (supplement) , S186-S187
- https://doi.org/10.1164/ajrccm.161.supplement_2.a1q4-8
Abstract
No abstract availableThis publication has 25 references indexed in Scilit:
- Overexpression of leukotriene C4 synthase in bronchial biopsies from patients with aspirin-intolerant asthma.Journal of Clinical Investigation, 1998
- Effect of a leukotriene B4 receptor antagonist, LY293111, on allergen induced responses in asthma.Thorax, 1996
- A controlled trial of the effect of the 5-lipoxygenase inhibitor, zileuton, on lung inflammation produced by segmental antigen challenge in human beingsJournal of Allergy and Clinical Immunology, 1996
- Exacerbations of asthma without sputum eosinophilia.Thorax, 1995
- Persistent increase in plasma and urinary leukotrienes after acute asthma.Archives of Disease in Childhood, 1995
- Single oral dose of prednisone decreases leukotriene B4 production by alveolar macrophages from patients with nocturnal asthma but not control subjects: Relationship to changes in cellular influx and FEV1Journal of Allergy and Clinical Immunology, 1994
- Leukotriene E4 and granulocytic infiltration into asthmatic airwaysThe Lancet, 1993
- Effect of cysteinyl-leukotriene receptor antagonist ICI 204.219 on allergen-induced bronchoconstriction and airway hyperreactivity in atopic subjectsThe Lancet, 1991
- Inhibition of Exercise-Induced Bronchoconstriction by MK-571, a Potent Leukotriene D4–Receptor AntagonistNew England Journal of Medicine, 1990
- Urinary leukotriene E4 levels during early and late asthmatic responses†Journal of Allergy and Clinical Immunology, 1990