A nonimprinted Prader–Willi Syndrome (PWS)-region gene regulates a different chromosomal domain in trans but the imprinted pws loci do not alter genome-wide mRNA levels

This publication has 54 references indexed in Scilit:

• Finishing the euchromatic sequence of the human genome
  Nature, 2004
• Cloning and characterization of a novel G-protein-coupled receptor with homology to galanin receptors
  Neuropharmacology, 2004
• A gene atlas of the mouse and human protein-encoding transcriptomes
  Proceedings of the National Academy of Sciences, 2004
• PGC-1α-responsive genes involved in oxidative phosphorylation are coordinately downregulated in human diabetes
  Nature Genetics, 2003
• Endocrine Dysfunction in Prader-Willi Syndrome: A Review with Special Reference to GH
  Endocrine Reviews, 2001
• Significance analysis of microarrays applied to the ionizing radiation response
  Proceedings of the National Academy of Sciences, 2001
• Chromosome Breakage in the Prader-Willi and Angelman Syndromes Involves Recombination between Large, Transcribed Repeats at Proximal and Distal Breakpoints
  American Journal of Human Genetics, 1999
• The human necdin gene, NDN, is maternally imprinted and located in the Prader-Willi syndrome chromosomal region
  Nature Genetics, 1997
• Expression of necdin, an embryonal carcinoma-derived nuclear protein, in developing mouse brain
  Developmental Brain Research, 1992
• A novel brain-specific mRNA encoding nuclear protein (necdin) expressed in neurally differentiated embryonal carcinoma cells
  Biochemical and Biophysical Research Communications, 1991