Inhibition of Ape1 nuclease activity by lead, iron, and cadmium.

This publication has 44 references indexed in Scilit:

• Elements in abasic site recognition by the major human and Escherichia coli apurinic/apyrimidinic endonucleases
  Nucleic Acids Research, 1998
• The crystal structure of the human DNA repair endonuclease HAP1 suggests the recognition of extra-helical deoxyribose at DNA abasic sites
  The EMBO Journal, 1997
• Sensitivity of Escherichia coli (MutT) and human (MTH1) 8-oxo-dGTPases to in vitro inhibition by the carcinogenic metals, nickel(II), copper(II), cobalt(II) and cadmium(II)
  Carcinogenesis: Integrative Cancer Research, 1997
• 3'-Phosphodiesterase activity of human apurinic/apyrimidinic endonuclease at DNA double-strand break ends
  Nucleic Acids Research, 1997
• The carcinogenicity of metals in humans
  Cancer Causes & Control, 1997
• The redox/DNA repair protein, Ref-1, is essential for early embryonic development in mice.
  Proceedings of the National Academy of Sciences, 1996
• Role of DNA repair inhibition in lead- and cadmium-induced genotoxicity: a review.
  Environmental Health Perspectives, 1994
• REPAIR OF OXIDATIVE DAMAGE TO DNA: Enzymology and Biology
  Annual Review of Biochemistry, 1994
• Removal of 3′-phosphoglycolate from DNA strand-break damage in an oligonucleotide substrate by recombinant human apurinic/apyrimidinic endonuclease 1
  Nucleic Acids Research, 1994
• Mutagenesis and comutagenesis by lead compounds
  Mutation Research/Genetic Toxicology, 1992