A Recurrent Network Involving the Transcription Factors PU.1 and Gfi1 Orchestrates Innate and Adaptive Immune Cell Fates

This publication has 46 references indexed in Scilit:

• Genome-wide Lineage-Specific Transcriptional Networks Underscore Ikaros-Dependent Lymphoid Priming in Hematopoietic Stem Cells
  Immunity, 2009
• Ebf1-mediated down-regulation of Id2 and Id3 is essential for specification of the B cell lineage
  Proceedings of the National Academy of Sciences, 2009
• E2A Proteins Promote Development of Lymphoid-Primed Multipotent Progenitors
  Immunity, 2008
• Regulation of B cell fate commitment and immunoglobulin heavy-chain gene rearrangements by Ikaros
  Nature Immunology, 2008
• Mutations in Growth Factor Independent-1 Associated with Human Neutropenia Block Murine Granulopoiesis through Colony Stimulating Factor-1
  Published by Elsevier ,2008
• The Transcriptional Repressor GFI-1 Antagonizes PU.1 Activity through Protein-Protein Interaction
  Journal of Biological Chemistry, 2007
• A Two-Step, PU.1-Dependent Mechanism for Developmentally Regulated Chromatin Remodeling and Transcription of the c-fms Gene
  Molecular and Cellular Biology, 2007
• Notch/Delta signaling constrains reengineering of pro-T cells by PU.1
  Proceedings of the National Academy of Sciences, 2006
• Gfi1:Green Fluorescent Protein Knock-in Mutant Reveals Differential Expression and Autoregulation of the Growth Factor Independence 1 (Gfi1) Gene during Lymphocyte Development
  Journal of Biological Chemistry, 2004
• Acute myeloid leukemia induced by graded reduction of a lineage-specific transcription factor, PU.1
  Nature Genetics, 2004