Activation of the IFN-Inducible Enzyme RNase L Causes Apoptosis of Animal Cells
- 1 September 1997
- Vol. 236 (2) , 354-363
- https://doi.org/10.1006/viro.1997.8719
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Inducible Expression of the 2-5A Synthetase/RNase L System Results in Inhibition of Vaccinia Virus ReplicationVirology, 1997
- 2-5A-dependent RNase Molecules Dimerize during Activation by 2-5AJournal of Biological Chemistry, 1995
- Identification of a novel serine/threonine kinase and a novel 15-kD protein as potential mediators of the gamma interferon-induced cell death.Genes & Development, 1995
- Temperature-Sensitive Mutants in the Vaccinia Virus A18R Gene Increase Double-Stranded RNA Synthesis as a Result of Aberrant Viral TranscriptionVirology, 1993
- Interferon‐γ arrests proliferation and causes apoptosis in stromal cell/interleukin‐7‐dependent normal murine pre‐B cell lines and clones in vitro, but does not induce differentiation to surface immunoglobulin‐positive B cellsEuropean Journal of Immunology, 1993
- The E3L gene of vaccinia virus encodes an inhibitor of the interferon-induced, double-stranded RNA-dependent protein kinase.Proceedings of the National Academy of Sciences, 1992
- A full-length murine 2-5A synthetase cDNA transfected in NIH-3T3 cells impairs EMCV but not VSV replicationVirology, 1990
- Modulation of ppp(A2'p)nA-dependent RNase by a temperature-sensitive mutant of vaccinia virusJournal of Virology, 1989
- Constitutive expression of (2′–5′) oligo A synthetase confers resistance to picornavirus infectionNature, 1987
- Eukaryotic transient-expression system based on recombinant vaccinia virus that synthesizes bacteriophage T7 RNA polymerase.Proceedings of the National Academy of Sciences, 1986