Glatiramer acetate-reactive T cells produce brain-derived neurotrophic factor
- 1 November 2003
- journal article
- Published by Elsevier in Journal of the Neurological Sciences
- Vol. 215 (1-2) , 37-44
- https://doi.org/10.1016/s0022-510x(03)00177-1
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Glatiramer acetate induces a Th2-biased response and crossreactivity with myelin basic protein in patients with MSMultiple Sclerosis Journal, 2001
- Glatiramer acetate in the treatment of multiple sclerosisExpert Opinion on Pharmacotherapy, 2001
- Mechanisms of action of glatiramer acetate in multiple sclerosisNeurology, 2001
- United States open-label glatiramer acetate extension trial for relapsing multiple sclerosis: MRI and clinical correlatesMultiple Sclerosis Journal, 2001
- Mechanisms of immunomodulation by glatiramer acetateNeurology, 2000
- Glatiramer acetate (Copolymer-1)-specific, human T cell lines: cytokine profile and suppression of T cell lines reactive against myelin basic proteinNeuroscience Letters, 2000
- Characterization of T cell lines derived from glatiramer-acetate-treated multiple sclerosis patientsJournal of Neuroimmunology, 2000
- Glatiramer acetate (Copaxone®) induces degenerate, Th2-polarized immune responses in patients with multiple sclerosisJournal of Clinical Investigation, 2000
- Treatment of multiple sclerosis with Copolymer-1 (Copaxone®): implicating mechanisms of Th1 to Th2/Th3 immune-deviationJournal of Neuroimmunology, 1998
- Copolymer 1 reduces relapse rate and improves disability in relapsing‐remitting multiple sclerosisNeurology, 1995