Dichloroacetate improves postischemic function of hypertrophied rat hearts
- 28 September 2000
- journal article
- research article
- Published by Elsevier in Journal of the American College of Cardiology
- Vol. 36 (4) , 1378-1385
- https://doi.org/10.1016/s0735-1097(00)00856-1
Abstract
No abstract availableKeywords
This publication has 23 references indexed in Scilit:
- Regression of Cardiac Hypertrophy Normalizes Glucose Metabolism and Left Ventricular Function During ReperfusionJournal of Molecular and Cellular Cardiology, 1997
- Regulation of myocardial carbohydrate metabolism under normal and ischaemic conditionsPotential for pharmacological interventionsPublished by Oxford University Press (OUP) ,1997
- Cardiac Efficiency Is Improved After Ischemia by Altering Both the Source and Fate of ProtonsCirculation Research, 1996
- Pyruvate Dehydrogenase Influences Postischemic Heart FunctionCirculation, 1995
- Calcium Overload during Reperfusion is Accelerated in Isolated Hypertrophied Rat HeartsJournal of Molecular and Cellular Cardiology, 1994
- Glucose fatty acid interactions and the regulation of glucose disposalJournal of Cellular Biochemistry, 1994
- Increased ischemic injury but decreased hypoxic injury in hypertrophied rat hearts.Circulation Research, 1990
- Tolerance of the hypertrophic heart to ischemia. Studies in compensated and failing dog hearts with pressure overload hypertrophy.Circulation, 1990
- Pyruvate‐enhanced phosphorylation potential and inotropism in normoxic and postischemic isolated working heartEuropean Journal of Biochemistry, 1989
- Fuel selection in animalsBiochemical Society Transactions, 1986