Mechanism of chronic obstructive uropathy: Increased expression of apoptosis-promoting molecules

This publication has 50 references indexed in Scilit:

• Down-regulation of Fas-Associated Phosphatase-1 (FAP-1) in Interleukin-2-Activated T Cells
  Cellular Immunology, 1998
• Agonistic anti-Fas antibodies induce glomerular cell apoptosis in mice in vivo
  Kidney International, 1997
• Apoptosis by Death Factor
  Cell, 1997
• Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell Death
  Cell, 1996
• TNF-Dependent Recruitment of the Protein Kinase RIP to the TNF Receptor-1 Signaling Complex
  Immunity, 1996
• Anti-Fas antibodies induce cytolysis and apoptosis in cultured human mesangial cells
  Kidney International, 1996
• The Novel Regulatory Mechanism of Fas System-Mediated Apoptosis in Mesangial Cells: Implication to Mesangial Proliferative Glomerulonephritis
  Biochemical and Biophysical Research Communications, 1996
• Mesangial cell apoptosis: the major mechanism for resolution of glomerular hypercellularity in experimental mesangial proliferative nephritis.
  Journal of Clinical Investigation, 1994
• The C. elegans cell death gene ced-3 encodes a protein similar to mammalian interleukin-1β-converting enzyme
  Cell, 1993
• A novel domain within the 55 kd TNF receptor signals cell death
  Cell, 1993