Effects ofGinkgo biloba extract (EGb 761) on gene expression: Possible relevance to neurological disorders and age-associated cognitive impairment
- 1 December 2002
- journal article
- research article
- Published by Wiley in Drug Development Research
- Vol. 57 (4) , 214-235
- https://doi.org/10.1002/ddr.10151
Abstract
No abstract availableKeywords
This publication has 99 references indexed in Scilit:
- Regulation of APP cleavage by α‐, β‐ and γ‐secretasesFEBS Letters, 2000
- Peripheral-type benzodiazepine receptors: Their implication in brain diseaseDrug Development Research, 2000
- Molecular interactions on microarraysNature Genetics, 1999
- Increased peripheral benzodiazepine binding sites in the brain of patients with Huntington's diseaseNeuroscience Letters, 1998
- Increased Activity‐Regulating and Neuroprotective Efficacy of α‐Secretase‐Derived Secreted Amyloid Precursor Protein Conferred by a C‐Terminal Heparin‐Binding DomainJournal of Neurochemistry, 1996
- Gene-Representing cDNA Clusters Defined by Hybridization of 57,419 Clones from Infant Brain Libraries with Short Oligonucleotide ProbesGenomics, 1996
- Excess EDRF/NO, a potentially deleterious condition that may be involved in accelerated atherogenesis and other chronic disease statesGeneral Pharmacology: The Vascular System, 1995
- Demonstration of the “anti-stress” activity of an extract of Ginkgo biloba (EGb 761) using a discrimination learning taskGeneral Pharmacology: The Vascular System, 1994
- The Nitric Oxide-Scavenging Properties of Ginkgo Biloba Extract EGb 761Biochemical and Biophysical Research Communications, 1994
- The physiology and mechanisms of the stress-induced changes in prolactin secretion in the ratLife Sciences, 1990