Zinc deficiency in rats decreases thrombin-stimulated platelet aggregation by lowering protein kinase C activity secondary to impaired calcium uptake
- 31 December 1995
- journal article
- Published by Elsevier in The Journal of Nutritional Biochemistry
- Vol. 6 (12) , 661-666
- https://doi.org/10.1016/0955-2863(95)00150-6
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Zinc status and distribution of protein kinase C in rat plateletsThe Journal of Nutritional Biochemistry, 1994
- Emptying of intracellular Ca2+ stores releases a novel small messenger that stimulates Ca2+ influxNature, 1993
- The role of protein kinase C in the initial events of platelet activation by thrombin assessed with a selective inhibitorThrombosis Research, 1993
- Rat platelets are deficient in internal Ca2+ release and require influx of extracellular Ca2+ for activationFEBS Letters, 1991
- Effects of dihydropyridines and inorganic calcium blockers on aggregation and on intracellular free calcium in plateletsBiochimica et Biophysica Acta (BBA) - Biomembranes, 1991
- Calcium Signaling in Human PlateletsAnnual Review of Physiology, 1990
- Differences in the effects of phorbol esters and diacylglycerols on protein kinase CBiochemistry, 1989
- Studies and Perspectives of Protein Kinase CScience, 1986
- Phorbol esters modulate thrombin-operated calcium mobilisation and dense granule release in human plateletsBiochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1986
- Activation of human platelets by ADP causes a rapid rise in cytosolic free calcium without hydrolysis of phosphatidylinositol-4,5-bisphosphateBiochemical and Biophysical Research Communications, 1985